W-18 was commonly reported to be an opioid in the popular press in the 2010s, which was later revealed not to be correct. W-18 was found to obtain weak activity at both sigma receptors and the translocator protein (peripheral benzodiazepinereceptor). It also inhibits hERG binding, an important antitarget in drug discovery, which possibly causes cardiovascular side-effects.
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In 2016 it was reported that Alberta had its first W-18 related death; however, toxicologists were unable to confirm that this product was the cause, as there were other drugs ingested by the individual. At this time, there have been no confirmed deaths in Alberta due to the drug W-18 exclusively. Currently, there are no standard screening tests for W-18 and in most cases, a strong suspicion would be needed to carry out specialized analysis. It is possible that W18 has been associated to other drug-related deaths by being added to similar effecting drugs, such as fentanyl.
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Other Names; W-1; W-32; 4-chloro-N-[1-[2-(4-nitrophenyl)ethyl]-2-piperidinylidene]-benzenesulfonamide, CAS 93101-02-1, W-15, beans, shady 80s, (4-chloro-N-[1-(2-phenylethyl)-2-piperidinylidene]-benzenesulfonamide),