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Pyrazolam is a drug of the benzodiazepine class. Benzodiazepine drugs contain a benzene ring fused to a diazepine ring, which is a seven membered ring with the two nitrogen constituents located at R₁ and R₄. Bromine is bound to this bicyclic structure at R₇. Additionally, at R₆ the bicylic core is substitued with a 2-pyridine ring.

What is Pirazolam?

Pyrazolam has structural similarities to alprazolam and bromazepam. Unlike other benzodiazepines, pyrazolam does not appear to undergo metabolism, instead being excreted unchanged in the urine. It is most selective for the α₂ and α₃ subtypes of the GABA_A receptor. Subjective effects include sedation, anxiety suppression, muscle relaxation, disinhibition, and euphoria. Pyrazolam is reported to produce comparably greater amounts of sedation and less euphoria than other benzodiazepines, limiting its recreational use.

How long does Pirazolam last in the body?

Pyrazolam also contains a 1-methylated triazole ring fused to and incorporating R₁ and R₂ of its diazepine ring. Pyrazolam belongs to a class of benzodiazepines containing this fused triazole ring, called triazolobenzodiazepines, distinguished by the suffix “-zolam“. Benzodiazepines are habit-forming and can produce mental and physical dependence with continued use. It is strongly advised to use harm reduction practices if using this substance.

What are the Effects of Pyrazolam?

Benzodiazepines produce a variety of effects by binding to the benzodiazepine receptor site and magnifying the efficiency and effects of the neurotransmitter gamma aminobutyric acid (GABA) by acting on its receptors. As this site is the most prolific inhibitory receptor set within the brain, its modulation results in the sedating (or calming effects) of pyrazolam on the nervous system.

How to use Pirazolam

Pyrazolam is most selective for the α2 and α3 receptor subtypes. It is excreted by the body unchanged thus not interacting with liver enzymes like other benzodiazepines, meaning that its use in people with reduced liver function may be safer. The anticonvulsant properties of benzodiazepines may be, in part or entirely, due to binding to voltage-dependent sodium channels rather than benzodiazepine receptors.

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